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What do we know about the new mutations?
An initial analysis of the new variant has been published and identifies 17 potentially important alterations.
There have been changes to the spike protein – this is the key the virus uses to unlock the doorway to our body’s cells.
One mutation called N501Y alters the most important part of the spike, known as the “receptor-binding domain”.
This is where the spike makes first contact with the surface of our body’s cells. Any changes that make it easier for the virus to get inside are likely to give it an edge.
The other mutation – a H69/V70 deletion, in which a small part of the spike is removed – has emerged several times before, including famously in infected mink.
Where has it come from?
The variant is unusually highly mutated.
The most likely explanation is the variant has emerged in a patient with a weakened immune system that was unable to beat the virus.
Instead their body became a breeding ground for the virus to mutate.
Does it make the infection more deadly?
There is no evidence to suggest that it does, although this will need to be monitored.
Will the vaccines work against the new variant?
Almost certainly yes, or at least for now.
All three leading vaccines develop an immune response against the existing spike, which is why the question comes up.
Vaccines train the immune system to attack several different parts of the virus, so even though part of the spike has mutated, the vaccines should still work.
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